![]() ![]() He has completed fellowship training in both intensive care medicine and emergency medicine, as well as post-graduate training in biochemistry, clinical toxicology, clinical epidemiology, and health professional education. He is an internationally recognised Clinician Educator with a passion for helping clinicians learn and for improving the clinical performance of individuals and collectives.Īfter finishing his medical degree at the University of Auckland, he continued post-graduate training in New Zealand as well as Australia’s Northern Territory, Perth and Melbourne. He is on the Board of Directors for the Intensive Care Foundation and is a First Part Examiner for the College of Intensive Care Medicine. He is also a Clinical Adjunct Associate Professor at Monash University. He is a co-founder of the Australia and New Zealand Clinician Educator Network (ANZCEN) and is the Lead for the ANZCEN Clinician Educator Incubator programme. thick skulls!)Ĭhris is an Intensivist and ECMO specialist at the Alfred ICU in Melbourne. ![]() difficult views in some patients (e.g.edema and vasospasm may be difficult to distinguish post-op.potential confounders include hypo/hypercapnia, haematocrit, BP.sensitivity 80% compared to angiography.can be used to monitor spasm post-treatment.reverberant flow (flow forward during systole and backward during diastole) = no sustainable cerebral perfusion pressure.increases in flow velocity may be vasospasm or hyperaemia (to differentiate compare flow through MCA with flow through ICA).the Lindegaard Ratio helps distinguish these conditions.high velocities in the MCA (>120cm/s) may be due to hyperaemia or vasospasm.Lindegaard Ratio = mean velocity in the MCA / mean velocity in ipsilateral extracranial internal carotid artery during vasospasm there is an increase in flow velocity through the narrowed segment that is proportional to the reduction in vessel diameter.phase shift is proportional to the speed of blood.probe on temporal bone -> measure flow in MCA. ![]()
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